ACADEMIC WRITING HELP FOR NURSING DEGREE

Academic writing helps for nursing degree

Academic writing helps for nursing degree

Haemolytic uraemic syndrome (HUS) is the most widespread cause of acute renal failure in children and is usually connected with Escherichia coli O157 infection. With a fatality rate of round 5%, some reports have affiliated antibiotic treatment with a worsening prognosis.

Description

We describe a child patient, initially treated for suspected meningococcal septicaemia, who went on to evolve renal complications and thrombocytopenia characteristic of HUS. Asubsequent affirmative stool experiment forE. coli O157 verified HUS as an appropriate diagnosis, although there were no clues of diarrhoea or vomiting all through the course of her management.

Feelings

Haemolytic uraemic syndrome (HUS) is a predominantly paediatric status that consists of the simultaneous triad of haemolytic anaemia, thrombocytopenia and acute renal failure. Although very rare (UK incidence: 0.71/100,000/yr [1]), it is the most widespread cause of acute renal failure in children, has a fatality rate of around 5% and is routinely connected with Escherichia coli O157 infection.

Evaluation

HUS is part of thrombotic microangiopathies that encompass Thrombotic thrombocytopenic purpura (TTP) and HUS. The classic childhood case of HUS happens after bloody diarrhea initiated by a strain ofE. coli that expresses verotoxin (also called Shiga-like toxin) which is renowned as STEC (Shiga toxin-producing E. coli) or EHEC (EnterohemorrhagicE. coli).(Collier, 2001) HUS follows an influenza-like or gastrointestinal (GI) prodrome with bleeding manifestations (especially hematemesis and melena), severe oliguria, hematuria, a microangiopathic hemolytic anemia, and (in some patients) famous neurologic changes.[4] A somewhat less widespread pattern of HUS (~10% of cases) does not pursue STEC contamination and is considered to result from factor H deficiency (a support regulatory protein) that results in uninhibited support activation after secondary endothelial wound resulting in thrombosis.

Analysis

In the classical pattern (90% of cases), the STEC toxin enters the bloodstream, attaches ...